非甾体类消炎药抑制结肠癌细胞株细胞生长的实验研究
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劳学军

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Experimental study on inhibition of cell growth in colon cancer cell lines by non-steroidal anti-flammatory drugs
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    摘要:

    目的:研究非甾体类消炎药(NASIDs)通过产生一氧化氮的亚硝基谷胱甘肽(GSNO)对结肠癌细胞生长的影响及其机制。方法:采用细胞生长曲线和流式细胞仪方法研究细胞凋亡,竞争酶联免疫分析测定细胞培养上清PGE2的环氧化酶2(PGE2)表达水平,Western Blot法检测COX-1/COX-2蛋白表达。结果:在不同时间和浓度梯度中,GSNO处理可增加PGE2的产量。用500μmol/L处理48h后,COX-1和COX-2的蛋白表达水平增加。NASIDs可以阻断PGE2的产生,但对于GSNO诱导的细胞生长抑制无影响。结论:GSNO可促进细胞产生PGE2增加,并诱导COX-1和COX-2蛋白呈时间依赖和剂量依赖性表达;高浓度的GSNO可抑制细胞生长,并诱导3种细胞株凋亡,而不受COX-2表达及其活性的影响。NADIs能阻断PEG2产生,但对于GSNO诱导的结肠癌细胞株生长抑制并无协同作用。

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    Abstract:Objective:To evaluate the effect and mechanism of non-steroidal anti-flammatory drags(NSAIDs) on the colon cancer cell growth by using S-nitrosoglutathione(GSNO) which can produce nitric oxide. Methods:Apoptosis of 3 colon cell lines were evaluated by cell growth curve and flow cytometry, the PGE2 levels in cell culture supernatants were determined by competitive enzyme immunoassay method, and the protein expression of COX-1 and COX-2 were analyzed by Western blot. Results:The production level of PGE2 was increased with CSNO treated concentration and time. Using 500μmol/L CSNO treatment for 48h, the expression level of COX-1 and COX-2 protein increased. NASIDs can block the production of PGE2 but had no effect on the inhibition of cell growth induced by GSNO.Conclusions:GSNO can increase PGE2 production and induce COX-1 and COX-2 protein expression in a dose-and time-dependent manner. Higher concentrations of GSNO also can inhibite cell growth and induced apoptosis in all 3 cell lines. NSAIDs can block production of PEG2 but NSAIDs are no effect on cell growth.

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劳学军,曹明溶,姜海平,汤汉林,梁忠平,庞钊.非甾体类消炎药抑制结肠癌细胞株细胞生长的实验研究[J].中国普通外科杂志,2006,15(2):7-110.
DOI:10.7659/j. issn.1005-6947.2006.02.007

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  • 在线发布日期: 2006-02-25